Linking Pathophysiology to Clinical Options in Psoriasis: New Insights into IL-23

Date: Friday 29 September 2017
Time: 12.30-14.00
Location: Mozart Hall 1–3

        

Faculty
Antonio Costanzo, Humanitas Clinical Research Center, Milan, Italy (Scientific Committee and Chair)
Carle Paul, Sabatier University, Toulouse, France (Scientific Committee)
Flavio Caprioli, University of Milan, Italy

Learning objectives

  • Describe the contributions of cytokines and autoantigens to the pathophysiology of psoriasis
  • Recognise the role of IL-23 in immune-mediated inflammatory diseases and analyse the similarities and differences between the pathophysiology of psoriasis, psoriatic arthritis and inflammatory bowel disease
  • Evaluate the key learnings from the latest clinical data and consider how this can be utilised to improve psoriasis treatment

Program

13.00-13.10 Welcome and introduction
Antonio Costanzo
13.10-13.30 The autoimmune story of psoriasis: A focus on autoantigens and cytokines
Antonio Costanzo
13.30-13.55 Lessons from IL-23 in immune-mediated inflammatory diseases
Flavio Caprioli
13.55-14.20 Linking pathophysiology and clinical evidence
Carle Paul
14.20-14.30 Interactive Q&A and discussion
Antonio Costanzo and Carle Paul

 With the support of Janssen Pharmaceutical Companies of Johnson & Johnson in EMEA.